the myth that cattle under 30 months of age are free from BSE/TSE is just that, a myth, and it's a false myth !
Information released on 2 February 2005 Summary of information requested What statistics are available on cattle less than 30 months of age found to have BSE? Information released VLA has recorded approximately 100 cases of BSE in cattle of 30 months of age or under during the entire period of the BSE epidemic (1986 - 2005). The figure is approximate as for 51 of these the age is only estimated. This is because farmers did not have accurate documentation to confirm birth date. This was not a requirement at the time. We can confirm that of the 100 cases, 49 were under 30 months of age, of these the youngest case was 20 months old.
http://www.defra.gov.uk/vla/vla/vla_ati_020205.htm
Youngest confirmed case 20 Months, Oldest confirmed case 22 Years, Data valid to 01 April 2008
http://www.defra.gov.uk/vla/science/docs/sci_tse_stats_gen.pdf
BSE Youngest and oldest cases by year of onset - GB 20 months, 21 months, (8) 24 months, see complete list of younger than 30 month ;
http://www.food.gov.uk/multimedia/pdfs/otmbsestatistics.pdf
BSE Youngest Japan 21 months, 23 months
http://www.jstage.jst.go.jp/article/ehpm/10/3/130/_pdf
The implications of the Swiss result for Britain, which has had the most BSE, are complex. Only cattle aged 30 months or younger are eaten in Britain, on the assumption, based on feeding trials, that cattle of that age, even if they were infected as calves, have not yet accumulated enough prions to be infectious. But the youngest cow to develop BSE on record in Britain was 20 months old, showing some are fast incubators. Models predict that 200-300 cattle under 30 months per year are infected with BSE and enter the food chain currently in Britain. Of these 3-5 could be fast incubators and carrying detectable quantities of prion.
http://www.sare.org/sanet-mg/archives/html-home/28-html/0359.html
Feed borne infection (31-34) a) Recent unpublished experiments at the VLA have shown that feeding exceptionally low doses (0.001g) of infected neural tissue can cause BSE. b) The working hypothesis of Defra that the major cause of BSE in BARBs cases has been through the ingestion of contaminated feed, most likely by young animals, is strongly supported. Thus control of the disease requires, as it has always required, completely eliminating the agent from the cattle feed chain. a) There has been a fall in the underlying incidence of BSE by birth cohort 1996/97 to 99/00 in GB, but the 2001/2 case leaves doubt subsequently. There has also been a fall in other countries except where feed controls were introduced later. 34. In view of the exceedingly low doses of brain material required to infect young cattle, the reductions in incidence consequent on the feed bans in the UK and elsewhere and the lack of evidence that other causes are responsible, the strongest hypothesis for BARBs is infection of animals via ingestion of BSE contaminated material.
http://www.defra.gov.uk/animalh/bse/pdf/hillreport.pdf
10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. MBM IN COMMERCE USA 2007
http://madcowfeed.blogspot.com/2008/04/substances-prohibited-from-use-in.html
MAD COW DISEASE terminology UK c-BSE (typical), atypical BSE H or L, and or Italian L-BASE
http://bse-atypical.blogspot.com/2008/03/mad-cow-disease-terminology-uk-c-bse.html
Sunday, April 20, 2008 Progress Report from the National Prion Disease Pathology Surveillance Center April 3, 2008
***Atypical forms of BSE have emerged which, although rare, appear to be more virulent than the classical BSE that causes vCJD.
see full text ;
http://prionunitusaupdate2008.blogspot.com/2008/04/progress-report-from-national-prion.html
CJD USA RISING
The statistical incidence of CJD cases in the United States has been revised to reflect that there is ONE CASE per 9000 in adults age 55 and older. Eighty-five percent of the cases are sporadic, meaning there is no known cause at present.
http://www.cjdfoundation.org/fact.html
Communicated by: Terry S. Singeltary Sr.
[In submitting these data, Terry S. Singeltary Sr. draws attention to the steady increase in the "type unknown" category, which, according to their definition, comprises cases in which vCJD could be excluded. The total of 26 cases for the current year (2007) is disturbing, possibly symptomatic of the circulation of novel agents. Characterization of these agents should be given a high priority. - Mod.CP]
http://pro-med.blogspot.com/2007/11/proahedr-prion-disease-update-2007-07.html
http://www.promedmail.org/pls/askus/f?p=2400:1001:6833194127530602005::NO::F2400_P1001_BACK_PAGE,F2400_P1001_PUB_MAIL_ID:1010,39963
There is a growing number of human CJD cases, and they were presented last week in San Francisco by Luigi Gambatti(?) from his CJD surveillance collection.
He estimates that it may be up to 14 or 15 persons which display selectively SPRPSC and practically no detected RPRPSC proteins.
http://www.fda.gov/ohrms/dockets/ac/06/transcripts/1006-4240t1.htm
http://www.fda.gov/ohrms/dockets/ac/06/transcripts/2006-4240t1.pdf
Sunday, April 20, 2008 Progress Report from the National Prion Disease Pathology Surveillance Center April 3, 2008
***Atypical forms of BSE have emerged which, although rare, appear to be more virulent than the classical BSE that causes vCJD.
see full text ;
http://prionunitusaupdate2008.blogspot.com/2008/04/progress-report-from-national-prion.html
Terry S. Singeltary Sr.
P.O. Box 42
Bacliff, Texas USA 77518
